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on 22-Nov-2016 (Tue)
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Parent (intermediate) annotation
Open ithe V 1 R has sites at both the amino-terminus and at the extracellular loop, whereas the V 2 R has a single site at the extracellular amino-terminus. [1] The well known antidiuretic effect of vasopressin occurs via activation of V 2 R. [1] <span>Vasopressin regulates water excretion from the kidney by increasing the osmotic water permeability of the renal collecting duct – an effect that is explained by coupling of the V 2 R with the G s signaling pathway, which activates cAMP. Interestingly, the V 2 R continues to activate G s after being internalize
Original toplevel document
Vasopressin receptor - Wikipedialuminal membrane of the collecting duct limit the antidiuretic action of vasopressin. Additionally, vasopressin selectively contracts efferent arterioles probably through the V 1 R, but not the afferent arteriole. [1] V 2 receptor[edit] <span>V 2 receptor (V 2 R) differs from V 1 R primarily in the number of sites susceptible to N-linked glycosylation; the V 1 R has sites at both the amino-terminus and at the extracellular loop, whereas the V 2 R has a single site at the extracellular amino-terminus. [1] The well known antidiuretic effect of vasopressin occurs via activation of V 2 R. [1] Vasopressin regulates water excretion from the kidney by increasing the osmotic water permeability of the renal collecting duct – an effect that is explained by coupling of the V 2 R with the G s signaling pathway, which activates cAMP. Interestingly, the V 2 R continues to activate G s after being internalized by β-arrestin rather than being desensitized. This internalized G s signaling by V 2 R is explained by the receptors ability to form "mega-complexes" consisting of a single V 2 R, β-arrestin, and heterotrimeric G s . [3] The increased intracellular cAMP in the kidney in turn triggers fusion of aquaporin-2-bearing vesicles with the apical plasma membrane of the collecting duct principal cells, increasing water reabsorption. [1] V 3 receptor[edit] The human V 3 receptor (V 3 R, previously known as V 1B R) is a G-protein-coupled pituitary receptor that, because of its scarcity, was only recently characteri
Flashcard 1421579521292
| status | not learned | measured difficulty | 37% [default] | last interval [days] | |||
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| repetition number in this series | 0 | memorised on | scheduled repetition | ||||
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Parent (intermediate) annotation
Open itCOP Jest to stosunek: ilości uzyskanej energii użytecznej (np. ciepła do ogrzewania), do energii włożonej – napędowej (np. energia elektryczna do zasilania sprężarki). COP (ang. coefficent of performance):
Original toplevel document (pdf)
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Parent (intermediate) annotation
Open itCo to jest ciepło ? Ciepło jest to forma wewnętrznej energii substancji. Substancje zawierają energię cieplną, aż do temperatury zera absolutnego (0 K = -273,15°C). Nawet powietrze o temperaturze -20°C zawiera ciepło, które mogą efektywnie wykorzystywać
Original toplevel document (pdf)
cannot see any pdfsFlashcard 1421581880588
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Parent (intermediate) annotation
Open itParowanie i skraplanie odbywa się przy stałym ciśnieniu
Original toplevel document (pdf)
cannot see any pdfsFlashcard 1421582929164
| status | not learned | measured difficulty | 37% [default] | last interval [days] | |||
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Parent (intermediate) annotation
Open itentalpia to zawartość energii
Original toplevel document (pdf)
cannot see any pdfsFlashcard 1421583977740
| status | not learned | measured difficulty | 37% [default] | last interval [days] | |||
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| repetition number in this series | 0 | memorised on | scheduled repetition | ||||
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Parent (intermediate) annotation
Open itentalpia to zawartość energii
Original toplevel document (pdf)
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Parent (intermediate) annotation
Open itIm mniejsza jest różnica temperatur pomiędzy temperaturą zasilania obiegu grzewczego i temperaturą wejściową źródła ciepła (∆T), tym wyższy jest współczynnik wydajności COP (efektywności). temperatura zasilania ogrzewania niższa o 1 K – COP wyższy o 2,5% temperatura źródła, np. solanki, wyższa o 1 K – COP wyższy o 2,7%
Original toplevel document (pdf)
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Queen Emma of Hawaii - Wikipedia
eleokalani (Anglican) House Kamehameha Father High Chief George Naʻea Thomas Rooke (hānai) Mother High Chiefess Fanny Kekelaokalani Young High Chiefess Grace Kamaʻikuʻi Young Rooke (hānai) Religion Church of Hawaii Signature [imagelink] <span>Emma Kalanikaumakaʻamano Kaleleonālani Naʻea Rooke of Hawaiʻi (January 2, 1836 – April 25, 1885) was queen consort of King Kamehameha IV from 1856 to his death in 1863. She ran for ruling monarch against King Kalākaua but was defeated. Cont
![[imagelink]](/wiki/File:Rookes1853.jpg){kind=link}
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Queen Emma of Hawaii - Wikipedia
talents as a vocalist, pianist and dancer were well known. She was also a skilled equestrian. Married life and reign[edit] [imagelink] Emma and Queen Victoria silver christening cup [imagelink] Emma and Kamehameha IV <span>Emma became engaged to the king of Hawaii, Alexander Liholiho. At the engagement party, a Hawaiian charged that Emma's Caucasian blood made her unfit to be the Hawaiian queen and her lineage was not suitable enough to be Alexander Liholiho's bride; she broke into tears and the king was infuriated. On June 19, 1856, she married Alexander Liholiho, who a year earlier had assumed the throne as Kamehameha IV. He was also fluent in both Hawaiian and English. Each nation and even the Chinese hosted balls and celebrations in honor of the newlyweds. Two years later on May 20, 1858 Emma gave birth to a son, Prince Albert Edward Kamehameha. During her reign, the queen tended palace affairs, including the expansion of the palace library. During her reign and after, she was known for her humanitarian efforts. Inspired by her adoptive father's work, she encouraged her husband to establish a public hospital to help the Native Hawaiians who were in decline due to foreign-borne diseases like smallpox. In 1859, Emma established Queen's Hospital and visited patients there almost daily whenever she was in residence in Honolulu. It is now called the Queen's Medical Center. Prince Albert, who was always called "Baby" by Emma, had been celebrated for days at his birth and every public appearance. Mary Allen, wife of the Chief Justice of the Supr
Flashcard 1421603638540
| status | not learned | measured difficulty | 37% [default] | last interval [days] | |||
|---|---|---|---|---|---|---|---|
| repetition number in this series | 0 | memorised on | scheduled repetition | ||||
| scheduled repetition interval | last repetition or drill |
Parent (intermediate) annotation
Open itVasopressin regulates water excretion from the kidney by increasing the osmotic water permeability of the renal collecting duct
Original toplevel document
Vasopressin receptor - Wikipedialuminal membrane of the collecting duct limit the antidiuretic action of vasopressin. Additionally, vasopressin selectively contracts efferent arterioles probably through the V 1 R, but not the afferent arteriole. [1] V 2 receptor[edit] <span>V 2 receptor (V 2 R) differs from V 1 R primarily in the number of sites susceptible to N-linked glycosylation; the V 1 R has sites at both the amino-terminus and at the extracellular loop, whereas the V 2 R has a single site at the extracellular amino-terminus. [1] The well known antidiuretic effect of vasopressin occurs via activation of V 2 R. [1] Vasopressin regulates water excretion from the kidney by increasing the osmotic water permeability of the renal collecting duct – an effect that is explained by coupling of the V 2 R with the G s signaling pathway, which activates cAMP. Interestingly, the V 2 R continues to activate G s after being internalized by β-arrestin rather than being desensitized. This internalized G s signaling by V 2 R is explained by the receptors ability to form "mega-complexes" consisting of a single V 2 R, β-arrestin, and heterotrimeric G s . [3] The increased intracellular cAMP in the kidney in turn triggers fusion of aquaporin-2-bearing vesicles with the apical plasma membrane of the collecting duct principal cells, increasing water reabsorption. [1] V 3 receptor[edit] The human V 3 receptor (V 3 R, previously known as V 1B R) is a G-protein-coupled pituitary receptor that, because of its scarcity, was only recently characteri
Flashcard 1421604687116
| status | not learned | measured difficulty | 37% [default] | last interval [days] | |||
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| repetition number in this series | 0 | memorised on | scheduled repetition | ||||
| scheduled repetition interval | last repetition or drill |
Parent (intermediate) annotation
Open itVasopressin regulates water excretion from the kidney by increasing the osmotic water permeability of the renal collecting duct
Original toplevel document
Vasopressin receptor - Wikipedialuminal membrane of the collecting duct limit the antidiuretic action of vasopressin. Additionally, vasopressin selectively contracts efferent arterioles probably through the V 1 R, but not the afferent arteriole. [1] V 2 receptor[edit] <span>V 2 receptor (V 2 R) differs from V 1 R primarily in the number of sites susceptible to N-linked glycosylation; the V 1 R has sites at both the amino-terminus and at the extracellular loop, whereas the V 2 R has a single site at the extracellular amino-terminus. [1] The well known antidiuretic effect of vasopressin occurs via activation of V 2 R. [1] Vasopressin regulates water excretion from the kidney by increasing the osmotic water permeability of the renal collecting duct – an effect that is explained by coupling of the V 2 R with the G s signaling pathway, which activates cAMP. Interestingly, the V 2 R continues to activate G s after being internalized by β-arrestin rather than being desensitized. This internalized G s signaling by V 2 R is explained by the receptors ability to form "mega-complexes" consisting of a single V 2 R, β-arrestin, and heterotrimeric G s . [3] The increased intracellular cAMP in the kidney in turn triggers fusion of aquaporin-2-bearing vesicles with the apical plasma membrane of the collecting duct principal cells, increasing water reabsorption. [1] V 3 receptor[edit] The human V 3 receptor (V 3 R, previously known as V 1B R) is a G-protein-coupled pituitary receptor that, because of its scarcity, was only recently characteri
Flashcard 1421606522124
| status | not learned | measured difficulty | 37% [default] | last interval [days] | |||
|---|---|---|---|---|---|---|---|
| repetition number in this series | 0 | memorised on | scheduled repetition | ||||
| scheduled repetition interval | last repetition or drill |
Parent (intermediate) annotation
Open itVasopressin regulates water excretion by increasing the water permeability of the collecting duct
Original toplevel document
Vasopressin receptor - Wikipedialuminal membrane of the collecting duct limit the antidiuretic action of vasopressin. Additionally, vasopressin selectively contracts efferent arterioles probably through the V 1 R, but not the afferent arteriole. [1] V 2 receptor[edit] <span>V 2 receptor (V 2 R) differs from V 1 R primarily in the number of sites susceptible to N-linked glycosylation; the V 1 R has sites at both the amino-terminus and at the extracellular loop, whereas the V 2 R has a single site at the extracellular amino-terminus. [1] The well known antidiuretic effect of vasopressin occurs via activation of V 2 R. [1] Vasopressin regulates water excretion from the kidney by increasing the osmotic water permeability of the renal collecting duct – an effect that is explained by coupling of the V 2 R with the G s signaling pathway, which activates cAMP. Interestingly, the V 2 R continues to activate G s after being internalized by β-arrestin rather than being desensitized. This internalized G s signaling by V 2 R is explained by the receptors ability to form "mega-complexes" consisting of a single V 2 R, β-arrestin, and heterotrimeric G s . [3] The increased intracellular cAMP in the kidney in turn triggers fusion of aquaporin-2-bearing vesicles with the apical plasma membrane of the collecting duct principal cells, increasing water reabsorption. [1] V 3 receptor[edit] The human V 3 receptor (V 3 R, previously known as V 1B R) is a G-protein-coupled pituitary receptor that, because of its scarcity, was only recently characteri
Flashcard 1421607570700
| status | not learned | measured difficulty | 37% [default] | last interval [days] | |||
|---|---|---|---|---|---|---|---|
| repetition number in this series | 0 | memorised on | scheduled repetition | ||||
| scheduled repetition interval | last repetition or drill |
Parent (intermediate) annotation
Open itVasopressin regulates water excretion by increasing the water permeability of the collecting duct
Original toplevel document
Vasopressin receptor - Wikipedialuminal membrane of the collecting duct limit the antidiuretic action of vasopressin. Additionally, vasopressin selectively contracts efferent arterioles probably through the V 1 R, but not the afferent arteriole. [1] V 2 receptor[edit] <span>V 2 receptor (V 2 R) differs from V 1 R primarily in the number of sites susceptible to N-linked glycosylation; the V 1 R has sites at both the amino-terminus and at the extracellular loop, whereas the V 2 R has a single site at the extracellular amino-terminus. [1] The well known antidiuretic effect of vasopressin occurs via activation of V 2 R. [1] Vasopressin regulates water excretion from the kidney by increasing the osmotic water permeability of the renal collecting duct – an effect that is explained by coupling of the V 2 R with the G s signaling pathway, which activates cAMP. Interestingly, the V 2 R continues to activate G s after being internalized by β-arrestin rather than being desensitized. This internalized G s signaling by V 2 R is explained by the receptors ability to form "mega-complexes" consisting of a single V 2 R, β-arrestin, and heterotrimeric G s . [3] The increased intracellular cAMP in the kidney in turn triggers fusion of aquaporin-2-bearing vesicles with the apical plasma membrane of the collecting duct principal cells, increasing water reabsorption. [1] V 3 receptor[edit] The human V 3 receptor (V 3 R, previously known as V 1B R) is a G-protein-coupled pituitary receptor that, because of its scarcity, was only recently characteri
Flashcard 1421608881420
| status | not learned | measured difficulty | 37% [default] | last interval [days] | |||
|---|---|---|---|---|---|---|---|
| repetition number in this series | 0 | memorised on | scheduled repetition | ||||
| scheduled repetition interval | last repetition or drill |
Parent (intermediate) annotation
Open itVasopressin regulates water excretion by increasing the water permeability of the collecting duct
Original toplevel document
Vasopressin receptor - Wikipedialuminal membrane of the collecting duct limit the antidiuretic action of vasopressin. Additionally, vasopressin selectively contracts efferent arterioles probably through the V 1 R, but not the afferent arteriole. [1] V 2 receptor[edit] <span>V 2 receptor (V 2 R) differs from V 1 R primarily in the number of sites susceptible to N-linked glycosylation; the V 1 R has sites at both the amino-terminus and at the extracellular loop, whereas the V 2 R has a single site at the extracellular amino-terminus. [1] The well known antidiuretic effect of vasopressin occurs via activation of V 2 R. [1] Vasopressin regulates water excretion from the kidney by increasing the osmotic water permeability of the renal collecting duct – an effect that is explained by coupling of the V 2 R with the G s signaling pathway, which activates cAMP. Interestingly, the V 2 R continues to activate G s after being internalized by β-arrestin rather than being desensitized. This internalized G s signaling by V 2 R is explained by the receptors ability to form "mega-complexes" consisting of a single V 2 R, β-arrestin, and heterotrimeric G s . [3] The increased intracellular cAMP in the kidney in turn triggers fusion of aquaporin-2-bearing vesicles with the apical plasma membrane of the collecting duct principal cells, increasing water reabsorption. [1] V 3 receptor[edit] The human V 3 receptor (V 3 R, previously known as V 1B R) is a G-protein-coupled pituitary receptor that, because of its scarcity, was only recently characteri
Flashcard 1421609929996
| status | not learned | measured difficulty | 37% [default] | last interval [days] | |||
|---|---|---|---|---|---|---|---|
| repetition number in this series | 0 | memorised on | scheduled repetition | ||||
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Parent (intermediate) annotation
Open itG-Beta-gamma part of the G protein of M 2 can open K + channels, which slows down the heart rate
Original toplevel document
Muscarinic acetylcholine receptor M2 - Wikipediais claim instead found no significant association between the CHRM2 gene and intelligence. [8] Olfactory behavior[edit] Mediating olfactory guided behaviors (e.g. odor discrimination, aggression, mating) [9] Mechanism of action[edit] <span>M 2 muscarinic receptors act via a G i type receptor, which causes a decrease in cAMP in the cell, generally leading to inhibitory-type effects. They appear to serve as autoreceptors. [10] In addition, they modulate muscarinic potassium channels. [11] [12] In the heart, this contributes to a decreased heart rate. They do so by the G beta gamma subunit of the G protein coupled to M 2 . This part of the G protein can open K + channels in the parasympathetic notches in the heart, which causes an outward current of potassium, which slows down the heart rate. Ligands[edit] Few highly selective M 2 agonists are available at present, although there are several non-selective muscarinic agonists that stimulate M 2 , and a number of selectiv
Flashcard 1421610978572
| status | not learned | measured difficulty | 37% [default] | last interval [days] | |||
|---|---|---|---|---|---|---|---|
| repetition number in this series | 0 | memorised on | scheduled repetition | ||||
| scheduled repetition interval | last repetition or drill |
Parent (intermediate) annotation
Open itVasopressin regulates water excretion by increasing the water permeability of the collecting duct
Original toplevel document
Vasopressin receptor - Wikipedialuminal membrane of the collecting duct limit the antidiuretic action of vasopressin. Additionally, vasopressin selectively contracts efferent arterioles probably through the V 1 R, but not the afferent arteriole. [1] V 2 receptor[edit] <span>V 2 receptor (V 2 R) differs from V 1 R primarily in the number of sites susceptible to N-linked glycosylation; the V 1 R has sites at both the amino-terminus and at the extracellular loop, whereas the V 2 R has a single site at the extracellular amino-terminus. [1] The well known antidiuretic effect of vasopressin occurs via activation of V 2 R. [1] Vasopressin regulates water excretion from the kidney by increasing the osmotic water permeability of the renal collecting duct – an effect that is explained by coupling of the V 2 R with the G s signaling pathway, which activates cAMP. Interestingly, the V 2 R continues to activate G s after being internalized by β-arrestin rather than being desensitized. This internalized G s signaling by V 2 R is explained by the receptors ability to form "mega-complexes" consisting of a single V 2 R, β-arrestin, and heterotrimeric G s . [3] The increased intracellular cAMP in the kidney in turn triggers fusion of aquaporin-2-bearing vesicles with the apical plasma membrane of the collecting duct principal cells, increasing water reabsorption. [1] V 3 receptor[edit] The human V 3 receptor (V 3 R, previously known as V 1B R) is a G-protein-coupled pituitary receptor that, because of its scarcity, was only recently characteri
Flashcard 1421612289292
| status | not learned | measured difficulty | 37% [default] | last interval [days] | |||
|---|---|---|---|---|---|---|---|
| repetition number in this series | 0 | memorised on | scheduled repetition | ||||
| scheduled repetition interval | last repetition or drill |
Parent (intermediate) annotation
Open itVasopressin regulates water excretion by increasing the water permeability of the collecting duct
Original toplevel document
Vasopressin receptor - Wikipedialuminal membrane of the collecting duct limit the antidiuretic action of vasopressin. Additionally, vasopressin selectively contracts efferent arterioles probably through the V 1 R, but not the afferent arteriole. [1] V 2 receptor[edit] <span>V 2 receptor (V 2 R) differs from V 1 R primarily in the number of sites susceptible to N-linked glycosylation; the V 1 R has sites at both the amino-terminus and at the extracellular loop, whereas the V 2 R has a single site at the extracellular amino-terminus. [1] The well known antidiuretic effect of vasopressin occurs via activation of V 2 R. [1] Vasopressin regulates water excretion from the kidney by increasing the osmotic water permeability of the renal collecting duct – an effect that is explained by coupling of the V 2 R with the G s signaling pathway, which activates cAMP. Interestingly, the V 2 R continues to activate G s after being internalized by β-arrestin rather than being desensitized. This internalized G s signaling by V 2 R is explained by the receptors ability to form "mega-complexes" consisting of a single V 2 R, β-arrestin, and heterotrimeric G s . [3] The increased intracellular cAMP in the kidney in turn triggers fusion of aquaporin-2-bearing vesicles with the apical plasma membrane of the collecting duct principal cells, increasing water reabsorption. [1] V 3 receptor[edit] The human V 3 receptor (V 3 R, previously known as V 1B R) is a G-protein-coupled pituitary receptor that, because of its scarcity, was only recently characteri